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Pediatr., 144:81-85
The possible neuroprotective actions of SGLT2 inhibitors are likely multifactorial
Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis
Research focus: initially explored in obesity and metabolic-syndrome models
In summary, the COMMIT trial suggests that beta-blocker therapy may be more appropriately started only after a patient has been stabilized hemodynamically post-MI, with the aim of preventing reinfarction and fibrillation
IGF-1 levels return to baseline within 2 weeks of stopping tesamorelin
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