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Thus, based on the clear and direct role of Y1-R in promoting glucose uptake in muscle, the lack of any response to GLP-1R agonism, and the glucose-reducing effects of GEP44 observed in our previous studies 16 , we predicted that GEP44 would also promote glucose uptake via interactions with the Y1-R

1,4 Hyperglycemia, dyslipidemia, and insulin resistance activate various metabolic pathways, resulting in oxidative stress, mitochondrial dysfunction, inflammation, and microvascular damage, culminating in neuronal injury, Schwann cell damage, and myelin sheath degeneration

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Eggs of liver flukes present oval sharp, with a convex operculum resting on the shoulders at the smaller end of the egg and a small knob at the opposite end (19)

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When deprived of adequate oxygen and nutrients, the central nervous system triggers an immediate and profound sensation of exhaustion, heavy limbs, and severe cognitive impairment, commonly referred to as "brain fog." Furthermore, this vascular dysregulation places an immense compensatory burden on the autonomic nervous system