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differentiation balance, a genetic screen was performed that identified mutations that result in a synthetic tumorous phenotype in gld-2(null) mutants ( pas-5 (proteasome alpha subunit), which encodes an -subunit of the 20S proteasome ( pas-5 partial-loss-of-function allele revealed that it likely affected the Notch signaling pathway, as well as the GLD-1 pathway functioning downstream of GLP-1/Notch signaling ( g ene) was found to be one of these proliferation promoting proteins, and is targeted for degradation by the E3 ubiquitin ligase RFP-1 (ring finger protein) ( glp-1(gf) background, suggesting that there could be multiple targets whose regulated degradation is necessary for the proliferation vs differentiation decision to be properly maintained (Table 1)

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