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Improves healing of wounds

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IGF1-LR3 targets insulin receptors and stimulates glucose uptake inside body cells, similar to all other analogs

Traditionally GLP-1(9-36) was considered inactive, however more recent studies indicate that GLP-1(9-36) can contribute to inhibiting glucagon secretion by inhibiting voltage-gated calcium channels through a GPCR mechanism, an effect which reduces the number of glucagon containing vesicles that are available for exocytosis, thereby contributing the the decrease in glucagon secretion (Figure 1)(Zheng et al, 2024)

and Thompson, L.U., 2004

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