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A major limitation of the present study is the cross-sectional design of the analyses, which estimated the associations between AC levels and depression at the same assessment (either baseline or 6-year follow-up), precluding conclusions about causality

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p38 upregulates E3 ligases (Atrogin-1, MuRF1, Nedd4) and autophagy genes (Atg7), while JNK phosphorylates c-Jun and FoxO to enhance their pro-atrophic activity (Changchien et al., 2019)

We don't yet understand how these drugs truly work What's the mechanism for the benefits of GLP-1 in the heart, liver or brain

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