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Over time, repeated oxidative injury promotes endothelial apoptosis and maladaptive repair responses, including smooth muscle proliferation and vascular calcification
Beyond the penumbra, the infarct region undergoes rapid necrosis, due to the lack of oxygen (Bandera et al., 2006), resulting in a hypoxic environment
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These diabetes-specific results mirror the pattern seen with CagriSema in REDEFINE 2, though direct comparison requires caution because the trial populations and designs differed
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