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Glutathione functions as a tripeptide antioxidant synthesised in the liver, scavenging reactive oxygen species and supporting Phase II detoxification enzymes

Its oral bioavailability is approximately 50%, and it reaches peak plasma concentration within 3 to 4 hours of dosing [1]

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demonstrated that in isolated rat pancreatic cells, the infusion of a highly selective SSTR2 antagonist entirely blocked the inhibitory effect on glucagon secretion, resulting in a swift increase in basal glucagon secretion levels

Decades of research and clinical use confirm minimal risk when used appropriately

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