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Cervicovaginal dysbiosis is thought to exacerbate this process by potentially facilitating HPV acquisition and persistence, and promoting viral transformation (including E6/E7 expression, viral integration, and genomic instability), and supporting secondary infections, such as those caused by Chlamydia trachomatis (Figure 3)
It is studied in controlled laboratory settings for research involving cellular recovery pathways, inflammatory-response signaling, extracellular matrix communication, tissue-remodeling models, angiogenic signaling, epithelial integrity, and peptide-driven repair mechanisms
There are over 100,000 glutathione citations on PubMed, and research continues worldwide
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