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ERR agonists, including SLU-PP-332 and SLU-PP-915, enhance mitochondrial oxidative capacity, promote FAO, reduce fibrosis, and preserve contractile function in preclinical studies, suggesting potential targeted metabolic therapies (Xu et al., 2024a)
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o Biocon Limited Advanced Pipeline Companies o Boehringer Ingelheim International GmbH o Amgen Inc
[25] Aller R, Izaola O, Gmez S, et al
Methylation pathways become saturated, and excess methionine is converted to homocysteine, which may contribute to cardiovascular risk if B-vitamin cofactors (B6, B12, folate) aren't adequate
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