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Without adequate VEGF signaling, wound healing stalls: fibroblasts deposit matrix without vascular support, creating avascular scar tissue rather than functional regeneration
In order to maintain intracellular glutamine-derived glutamate levels, NRF2-addicted cells exploit glutamine transporter, SLC1A5, to uptake more extracellular glutamine that is then converted to glutamate by glutaminase (Romero et al
Zhang L, Luo YL, Xiang Y, Bai XY, Qiang RR, Zhang X, et al
Kushner RF, Shapiro M
The strains S
Early clinical studies have shown encouraging results, but it is still being evaluated
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