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The Science: How BPC-157 Works Mechanism 1: Angiogenesis (Blood Vessel Growth) BPC-157's primary mechanism appears to be stimulating new blood vessel formation: Upregulates VEGF (Vascular Endothelial Growth Factor) Enhances growth factor production at injury sites Improves blood flow to healing tissue Better nutrition and oxygen delivery to repair mechanisms This is particularly important for tendon recovery because improved vascularization directly correlates with faster healing
Seizure-induced reduction in glucose utilization promotes brain hypometabolism during epileptogenesis
Similarly, gene mutations or problems in the production of the other hormones of the somatotropic axis and their receptors can cause a domino effect
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Another interesting outcome was that GSH treatment did not change the efficiency of MRAB resistance to the antibiotics (Augmentin, ciprofloxacin, gentamicin and tobramycin) tested in this study (Supplementary Figures S1AD), indicating that GSH does not influence MRABs gene expression profile, however, further research is needed to ascertain the validity of these findings
