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Research Background & Mechanistic Focus Current research use centers on coordinated cytoskeletal, cellular-migration, and structural-response signaling models, comparative presentation or formulation behavior where applicable, and controlled experimental models designed to evaluate compound-specific signaling activity within non-clinical laboratory systems

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Another link between protein misfolding and redox mechanisms is illustrated by the presence of important redox proteins in the misfolded protein inclusions in ALS

This conclusion is based on showing that L-carnitine suppressed the following TRPV1-induced responses: (1) 1 mmol/l L-carnitine reduced CAP-induced increases of the whole-cell currents (Fig