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While immunotherapy has revolutionized the treatment of many solid tumors, its application to osteosarcoma has yielded modest clinical benefits, primarily because these tumors are immunologically cold (57)

Our study indicates that upregulation of p53-pS15 and p16 maintains a persistent senescent microenvironment to promote cell cycle arrest and apoptosis resistance in keloid fibroblasts

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Adequate protein maximizes IGF-1 LR3 benefits

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