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Ligand binding to TLRs typically recruits the adaptor protein myeloid differentiation primary response 88 (MyD88), thereby triggering major pro-inflammatory signaling cascades, including the MAPK and nuclear factor kappa B (NF-B) pathways (165, 259)

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References Chen Y, Fan Z, Yang Y, Gu C

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Additionally, beyond its apparent influence on growth hormone synthesis, researchers state that CJC-1295 [apparently] caused an increase in total pituitary RNA and GH mRNA, suggesting that proliferation of somatotroph cells had occurred, as [supported] by immunohistochemistry images, suggesting possible enhancement in the cellular machinery responsible for GH production

Yaghoubi, N