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Because the beta-oxidation of fatty acids requires substantial amounts of oxygen to proceed, this hypoxic environment further stalls the mitochondrial engines
As evident in fig
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The abovementioned process activates the Nod1/NF-kB/MAPK/ERK/FOXO4 signaling pathway, thereby disrupting the programmed apoptosis of the cells, which is regarded as a protective mechanism in the organisms
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