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By demonstrating that the experimentally impaired SYK function exacerbates pathology, our work underscores the protective role of SYK in immune-mediated peripheral neuropathy and its potential as a therapeutic target
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Tesofensine becomes valuable when the bigger issue is metabolic slowdown, fatigue, or plateauing after initial GLP-1 success
It also has an importance for the psyche
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J Cutan Pathol
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