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Due to (temporarily) reduced sodium reuptake while continuously promoting glucosuria, these drugs induce a reduction in GFR likely via activation of tubuloglomerular feedback (TGF) that may represent reduced intraglomerular pressure [5]
In the context of obesity, the accumulation of free fatty acids in joints leads to organ lipotoxicity, and rising lipid levels contribute to the occurrence of KOA, with consequent tissue inflammation [12]
It is intended strictly for in-vitro laboratory research use only
non-senescent cells
It does not authorize unrestricted manufacturing or mass-market sales
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