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10.3389/fnut.2023.1267035 167
doi: 10.1136/annrheumdis-2013-204273 7 DalbethNPhipps-GreenAFramptonCNeogiTTaylorWJMerrimanTR
G.JiangT
On the other hand, its high reactivity also contributes to the uncontrolled removal of electrons, which underlies pathological cell damage through the propagation of highly reactive oxygen-containing molecules
Reduced forms, like NMNH, are created when molecules gain electrons, which can change how they work or are used
Our results showed that inhibiting ferroptosis reduced the release of profibrotic cytokines from TECs and suppressed the TGF-/Smad signaling pathway during fibrosis
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