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S -propenyl cysteine, a derivative, can inhibit the NF-B pathway and activate the Keap1/Nrf2 pathway, mitigating inflammation and oxidative stress, thereby supporting cysteine's role in managing autoimmune disorders (Mo et al
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Regulation of hypoxia-inducible factor 1 is mediated by an O 2 -dependent degradation domain via the ubiquitin-proteasome pathway
In addition, the healed tendons are usually weaker than before injury
ATAD3B is a mitophagy receptor mediating clearance of oxidative stress-induced damaged mitochondrial DNA
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