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In an environment characterized by oxidative stress and mitochondrial dysfunction, the presence of oxLDL further increases the secretion of proinflammatory cytokines (e.g., tumor necrosis factor , interleukin [IL]-6 and IL-1) and the expression of adhesion molecules (including vascular cell adhesion protein [VCAM]-1, monocyte chemoattractant protein [MCP]-1, intercellular adhesion molecule [ICAM]-1 and E-selectin) by monocytes and macrophages ( GLP-1 receptor stimulation appears to attenuate these processes in preclinical models and human studies in various ways
Comprehensive inflammation control: Both peptides reduce inflammation through different mechanisms, giving broad control without impairing healing
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However, cysteine residues of peroxiredoxins are easily hyperoxidized to cysteine sulfinic acid, resulting in the inactivation of peroxidase activity
Endocr Metab Immune Disord Drug Targets 2025;25:267-70
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