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When IGF-IR is deleted in mature osteoblasts, using OCN promoter-driven Cre line, the KO mice produce less trabecular bone, with an increased proportion of osteoid in the bone, indicating impaired mineralizing functions in OCN IGF-IR / osteoblasts (58,59)

Acknowledgements This work was supported by the National Natural Science Foundation of China [grant numbers 82125037], Science Foundation for Distinguished Young Scholars of Jiangsu province [grant number BK20231528], Key R&D Program of Jiangsu Province [grant number BE2022815], Jiangsu Provincial Medical Innovation Center [grant number CXZX202225], Young Elite Scientists Sponsorship Program of the China Association for Science and Technology [grant number 2021-QNRC2-B17], and 333 high-level talents project of Jiangsu Province for Yang Yang [grant number BRA202202]

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We utilized Students t-tests and Pearson correlations for parameter comparisons

Recently, a study demonstrated that ferroptosis may be involved in the occurrence of AD by using bioinformatics analysis[56], while our present study confirmed that ferroptosis of SMCs is an important cause of medial degeneration of the aorta and the progression of AD by conducting experiments in HASMCs, mice and specimens of patients with AD

Aging and age-related diseases Aging is a universal, intrinsic, irreversible, heterogeneous and multidimensional process of progressive involution characterized by a gradual loss of physiological functions that increases the probability of death