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Taken together, these results suggest that Gln-derived aKG is an important cofactor required for histone modification and that decreased Gln-derived aKG production suppresses histone demethylation, specifically H3K9me3 demethylation, by inhibiting KDM4C activity, leading to significant downregulation of cFLIP expression
The body efficiently regulates copper through hepatic metabolism and biliary excretion
In terms of IL-1 production, LPS-stimulated BV-2 cells pretreated with 100 and 1000 M of -EC or GSH showed a significant reduction compared to the control (Fig
2018;496(2):648-653.[DOI] 102
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