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Front Neurosci 15: 781964
Wu D, Chen C, Katoh K, Zhang J, Clarke IJ
We thus tried to investigate the potential for VRK2 phosphorylating Sec24C and found that the phosphorylation level of Sec24C was attenuated in VRK2-KO cells but elevated when restoring the expression of VRK2 (Fig
One of the reasons for this is that it requires a large amount of oxygen to function normally, which as a consequence leads to a large production of free radicals , which is why our antioxidant status is vital in mental health and neurological conditions
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