Interestingly, hyperplasia of selected EEC subtypes in response to intestinal infection is probably dependent on immune cells, since serotonin (5-HT)-producing cell hyperplasia associated with Citrobacter rodentium infection was absent in mice lacking adaptive immunity ( + T cells were necessary to increase numbers of 5-HT producing enterochromaffin cells during helminth infection (149)
10.3892/ol.2012.931 Summary Keywords Alzheimers disease, glutathione, multiple sclerosis, Parkinsons disease, redox imbalance Citation Knudsen E, Tadje J, Coggins C and Venketaraman V (2026) Glutathione and neurodegenerative diseases: immunopharmacological implications
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