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Neuroendocrine Modulation: Influences pituitary release of GH, LH, and ACTH [Kastin 1990]

Journal of Experimental Pharmacology, 2012

McGuire F.P

This unique clinical-molecular feature suggests that dysregulation of the TP53 signaling pathway is not only a key pathogenic mechanism of AML, but also a potential marker for molecular stratification and a target for targeted therapies, supporting the optimization of precision treatment strategies (12)

Leydig cell aging (Zhang et al., 2020, Aging) FOXO4-DRI improved testosterone secretion and the testicular microenvironment in aged mice by clearing senescent Leydig cells

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