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Furthermore, there arent any plausible pharmacological mechanisms to support such effects

Wen Q, Zhang L, Zhao F, Chen Y, Su Y, Zhang X

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If SAH accumulates, homocysteine is poorly managed or sulfur-amino-acid substrate is inadequate, the liver may have less flexibility to maintain methylation while also supplying cysteine for glutathione and taurine

This delivery method can provide faster systemic availability compared to oral supplements, which may lose potency during digestion

This is presumably due to the fact that mitochondria do not have a GSH synthesizing enzyme system and depend upon the energy/transporter-dependent transfer of GSH from the cytosol [37]