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TGF-1 has a neuroprotective effect by inhibiting microglial activation via inflammatory signals, such as IFN- and MPP+ toxicity [209, 210]

Although some authors have tried to assess the mechanistic link between HLA-II allele expression and the development of ME/CFS, the lack of robust data makes it difficult to support this association (88, 490)

Data from cell culture experiments such as those by Maar et al

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By targeting miR-223-3p/ITPR3/GPX4/xCT, the researchers successfully prevented high glucose-induced ferroptosis [123] (Table 2)

Subsequently, the 17 identified drug-disease targets were uploaded to the STRING database