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Additionally, obesity and IR both contribute to -cell dysfunction and apoptosis, mechanistically explained by glucotoxicity and increased immunogenicity (the accelerator hypothesis) or by -cell exhaustion due to increased metabolic demand (the overload hypothesis) [23]

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Secretagogin is highly expressed in enteroendocrine K cells and plays a critical role in nutrient-induced GIP secretion

This next-generation agent (inspired by Lillys triple agonist concept) is intended for treatment-resistant obesity and fatty liver reduction ( [41] )