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Given the extensive evidence linking redox dysfunction to ALS, it is not surprising that several redox-active molecules have been trialed as potential therapeutic agents
The protective effects of GIP on the bone have been also observed in humans with and without diabetes [43, 44]
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Semaglutide belongs to this group, and its primary action involves mimicking the effects of a naturally occurring hormone called glucagon-like peptide-1 (GLP-1)
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