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252 BCG and placebo responders were followed for 17 months

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Unlike BPC 157, which primarily works through growth factor signaling pathways, TB 500 operates by binding G-actin to facilitate cellular motility particularly relevant in wound healing, cardiac repair, and musculoskeletal injury models

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The affected pathways clustered into several categories relevant beyond cosmetic applications: Extracellular matrix remodeling: Upregulation of decorin, multiple collagen types (I, III, VII), and matrix metalloproteinase inhibitors (TIMP-1, TIMP-2)