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All randomized participants received at least one dose of the study drug and were included in both the full and safety analysis populations [1]
Prodrugs per se are therapeutically inactive (73), undergo enzymatic and/or chemical bioconversion, and release active parent drug at the target site within the brain
Notably, the N -linked glycosylation status of DAT influences its cell surface expression and transporter activity (Hastrup et al., 2001
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Moreover, the improvement of pharmacological effect in disease models in vivo by prodrug approach was shown only for the prodrug of 7-chlorokynurenic acid (133, 134) and ibuprofen prodrugs (145) when compared to parent drug administration
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