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HIV-1 protein Vpr increases H 2 O 2 production and activates hypoxia-inducible factor-1, thereby stimulating HIV-1 gene transcription via hypoxia-inducible factor-1 association with the HIV-1 long terminal repeats 122

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For BV-2 cells pretreated with -EC (1, 10, 100, and 1000 M), a significant reduction in LPS-induced NO production was observed specifically in cells treated with 100 M -EC compared to the LPS-stimulated control cells (Fig