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The clinical consequence of an adverse event (major bleeding in an anticoagulated patient) is high severity

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Through literature review, it is speculated that in animal models of neurodegenerative diseases, depletion of long-term nuclear receptor-assisted activator (NCOA4) in the brain can worsen the neurodegenerative disease phenotype due to the further inappropriate accumulation of free iron and resulted in oxidative stress (Quiles Del Rey and Mancias, 2019), and then induced ferroptosis in nerve cells

The selectivity data showing absence of ACTH/cortisol stimulation come from swine studies, and while swine GH physiology closely resembles that of humans, direct translation cannot be assumed without clinical validation

These results help understand the methodological practicability of ultra-filtration to wash out un-reacted LASSS from the LASSS-treated HGF specimens