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Unlike TB-500 (Thymosin Beta-4 fragment), which acts primarily via G-actin sequestration and endothelial progenitor cell mobilization, BPC-157 exerts its action via an NO-dependent pathway and VEGFR2 modulation two distinct but potentially complementary mechanisms, which is why research protocols sometimes combine both peptides (synergy documented in animal tendon repair models)
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10.2174/13816128113190990411 78 SikiricP.SeiwerthS.RucmanR.TurkovicB.RokotovD
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Therefore, the studies to decipher the contributing factors on the distribution of nasally administered molecules in blood and the brain are necessary for the desirable and safe applications
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