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e , NCI-H716 cells were treated with GPCR antagonists (10 M) or vehicle (DMSO) for 15 min before P9 treatment (50 g ml 1 ), then incubated for an additional 2 h, after which GLP-1 secretion was analyzed (n = 6 per group)

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Pharmacological activation of the GLP-1 Receptor has also shown to help exogenous insulin in the control of glycaemia in patients with type 1 diabetes, by slowing the gastric emptying and blocking glucagon secretion (183, 184)

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