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FIGURE 3 In summary, this evidence suggests that the origin of excess ROS production is the result of (1) defective mitochondria (due to the abnormal and reduced distribution of cardiolipins) with high intermembrane electrical potential and (2) reduced mitophagy process (due to high expression and activity of P53 protein)

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While cancer cells typically evade apoptosis at this stage under untreated conditions, BRs are capable of triggering apoptotic processes during the G1 phase (302)

Efficacy of Antioxidant Supplementation on Conventional and Advanced Sperm Function Tests in Patients with Idiopathic Male Infertility

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