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These cells significantly influence immune suppression in cancer, contributing to tumor vascular development, therapy resistance, and increased metastasis, all of which are well supported by existing evidences

These mechanisms are worth mentioning since if the results in this latest correspondence in the NEJM are indeed replicated, there is a large difference if these effects are occurring either a) Because pancreatic beta cells are dying less b) Because pancreatic beta cells are growing back c) Because pancreatic beta cells are not being targeted by the immune system anymore If replicated, I find a) and b) to be the most likely reasons

It remains an unapproved research compound with significant safety concerns

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