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Therefore, loss of ETF:QO (encoded by ETFDH ) from the mitochondria as revealed by the proteomic analysis may be part of the explanation for the MADD-like changes in addition to the profound deficiency of Complex I

NAC, a direct antioxidant and GSH precursor, inhibits NF-B and other cytokines that contribute to inflammation, which results in the downregulation of many pro-inflammatory genes

Limited nutrient availability in the tumor microenvironment renders pancreatic tumors sensitive to allosteric IDH1 inhibitors

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