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Nikoo S, Ebtekar M, Jeddi-Tehrani M, et al

P., & Nagaraju, K

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In cultured pulmonary endothelial cells in vitro, carnitine can effectively reduce the apoptosis, damage to migration and obstruction of angiogenesis caused by hyperoxia, resulting in improvements in endothelial cell dysfunction and persistent lung injury under hyperoxic conditions [308]

Here, we employed two different experimental settings in which glutathione and NADP redox responses are diametrically opposed, to test whether NERNST responds predominantly to NADP or glutathione redox changes