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Phase 2a trial of GSBR-1290 (45mg and 90mg) in 54 T2DM patients over 12 weeks observed significant reduction in HbA1c (45 mg: -1.01%, p=0.008

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It activates PI3K/Akt and MAPK pathways, preventing neuronal death caused by amyloidogenesis, glucose deprivation, neuroinflammation, and apoptosis (8)

Emerging data suggest that disruptions in the glutathione redox system play a crucial role in the pathophysiology of autism