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doi: 10.1016/j.molmed.2018.01.001 80 WuBWangKFeiJBaoYWangXSongZet al

It works by binding to GHRH receptors in the pituitary gland, stimulating the pulsatile release of endogenous growth hormone (GH)

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69 Conversely, the extracellular release of decorin (DCN, an extracellular matrix-derived damage-associated signaling proteoglycan), 70 high mobility group box 1 (HMGB1, an architectural DNA-binding factor in the cell nucleus), 71 and nuclear receptor coactivator 4 (NCOA4, a mediator of ferritinophagy) 72,73 may function as damage-associated alarm signals that promote inflammatory responses in the local microenvironment

Furthermore, pro-inflammatory and pro-fibrotic cytokines, including activin A, visfatin, transforming growth factor-1 (TGF-1), and MCP-1, promote fibroblast proliferation and myofibroblast activation, resulting in atrial and ventricular arrhythmias (103)