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Abstract The function and physiology of the central nervous system (CNS) can be affected by of bacterial, fungal, protozoan, and viral infections
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Lee AJ et al
(106) used CoQ10-encoded liposome/gold hybrid nanoparticles to attenuate RA progression by targeting signal transducer and activator of transcription-3/T helper cell 17 (STAT3/Th17)
All of these findings suggest that TRPV4 stimulation generates oxidative neurotoxicity, neuropathic pain, and apoptosis in the STZ-induced diabetic mice
These death receptors bear a death domain (DD) that fosters intracellular proteinprotein interactions, which are pivotal in transmitting apoptosis-inducing signals
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