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Therefore, therapeutic agents capable of mitigating oxidative damage are of significant interest
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Compared to changes in species abundance, more discrete effects were observed on associated KEGG modules, which is likely explained by redundancy of functional metabolic features among regulated bacterial species
However, NAC is likely ineffective in increasing GSH levels in cells with dysfunctional GCL, in which -EC synthesis from Cys converted from NAC would be suppressed [5]
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