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Interestingly, evidence from metabolomic studies has shown associations between abnormal acylcarnitine and acetylcarnitine levels in several of the aforementioned fatigue-associated neuropsychiatric diseases, as reviewed elsewhere [29]

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The fact that this polymorphic enzyme partially regulates metabolic disposition leads us to postulate that acute toxicity, drug abuse and dependence as well as, in some cases, long-term neurotoxicity could be influenced by CYP2D6 genetics (Sellers and Tyndale, 2000

0.1 mL delivers the 0.35 mg target