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These changes were mirrored by changes in the lung (Figure 5A) and liver (Figure 5B) tissue GCL activity which is a good indicator of a tissue's capacity to maintain GSH levels

The source data for all figures are provided in Supplementary Data 1

These positive results indicate that the change in myocardial cells may not be the primary driver of HFpEF

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Furthermore, pre-synaptic and post-synaptic proteins, synaptophysin and PSD-95, which reflect synaptic disruption [122], were upregulated, neuronal excitability normalized, and intracellular calcium, a key regulator in metabolic dysfunction and progressive neuronal loss [123], was modulated [121]