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DA-CH5 reduced amyloid plaques and phosphorylated tau [112], while DA-CH3 reduced amyloid plaque as well as resolving endoplasmic reticulum stress and derailed autophagy [111]
At worst, it could produce unintended and adverse effects
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These pathways may involve both receptor-dependent and receptor-independent mechanisms related to metabolic regulation
Novo Nordisk is doubling down on cardiometabolic diseases, expanding semaglutide into additional indications while also developing next-generation oral GLP-1 candidates
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