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Patients who have early Leber's disease (hereditary optic nerve atrophy) have been found to suffer severe and swift optic atrophy when treated with vitamin B12
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The goal is to relieve deficiency-related fatigue, not to provide a stimulant-like boost in people who are not deficient
By restoring cellular NAD+ levels and optimizing mitochondrial output, providers can help patients overcome the biochemical barriers that prevent fat oxidation
The injectable route ensures adequate B12 delivery regardless of gut function
Clinical Applications: Who Is This For
