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Given pulmonary surfactants pivotal role in RDS pathophysiology and the dependency of palmitoyl-carnitine synthesis on adequate L-carnitine availability, strategies to augment L-carnitine levels represent a biologically plausible approach to support surfactant production and potentially ameliorate RDS severity
NO represses the oxygenation of arachidonoyl PE by 15LOX/PEBP1: mechanism and role in ferroptosis
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One study in the US found elevated levels of mercury and other toxic metals in the blood [6]
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