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Peptides occupy a central position in AD pathobiology: pathogenic species such as A oligomers and tau-derived fragments drive synaptic failure, mitochondrial dysfunction, and neuroinflammation, whereas several endogenous neuropeptides exert compensatory neuroprotective, trophic, or homeostatic effects

Sensitivity analyses, including MR-Egger and leave-one-out tests, demonstrated consistent effect estimates across methods, further supporting the reliability of our results

Glutathione works in two phases to reduce the damaging and health-threatening effects of oxidation

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