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The TB-500-exposed tissues also featured more uniform spacing between fibrils, suggesting a more orderly matrix that may support stronger tissue formation
Crucially, interactions between NSW and specific polymorphisms in CRY2 and RORA further elevated BC risk, underscoring the complex interplay of genetic susceptibility and environmental circadian disruption in BC etiology 195
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4.2 Detailed expandable studies Study 1: Lau 2021, pivotal Phase 2 monotherapy Citation: Lau D.C.W., Erichsen L., Francisco A.M., et al
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